I-X Factor Ichaza i-Androgyny kwi-Male Asperger’s

-Delile

• I-Asperger's Syndrome Essential Reads
• Iseluleko Somshado Samahhala Esivela Kubantu Abadala baka-Asperger

Njengoba ucwaningo lwamuva lukhomba, “Umbono‘ wobuchopho besilisa obedlulele ’usikisela ukuthi i-autism spectrum disorder (ASD) ihluke ngokweqile kobuhlakani besilisa. Kodwa-ke, kuyindida, ukuthi abantu abaningi abane-ASD babonisa izici zomzimba eziyingozi ngaphandle kobulili. ”

Izithombe zobuso nezomzimba, kanye nokuqoshwa kwezwi, zitholwe futhi zahlolwa maqondana nokuhlangana kobulili, ngokungaboni nangaphandle, ngabahloli abayisishiyagalombili. I-psychiatric symptomatology, amazinga ama-hormone, i-anthropometry, kanye nesilinganiso sobude be-2 kuya ku-4 (2D: 4D, kwesobunxele) kukalwe kubantu abadala abangama-50 abane-ASD esebenza kahle kanye nobudala be-53- kanye nezilawuli ze-neurotypical ezihambelana nobulili.

Ubude obuhlobene beminwe buqondiswa ukukhulelwa kwamasonto ayi-14, futhi kukhombisa amathonya e-hormonal. Emadodeni, umunwe wendandatho (4D) uvame ukuba mude kunomunwe wenkomba (2D), kepha lesi silinganiso sithambekele ekulinganeni kwabesifazane. Ucwaningo lwangaphambilini luthole ukuthi isilinganiso esiphakeme esihambisana nobufazi, umdlavuza webele, kanye nobuningi besifazane / obuphansi besilisa. Isilinganiso esiphansi esihambisana nobudoda, isandla sobunxele, ikhono lomculo ne-autism. Kodwa-ke, lolu cwaningo luthole ukuthi abesilisa abaseqenjini le-ASD "babonisa ngaphezulu (okusho ukuthi abesilisa abancane) 2D: izilinganiso ze-4D, kodwa amazinga afanayo we-testosterone kuzilawuli."

Ababhali babika ukuthi abesifazane abane-ASD babenamazinga aphakeme futhi aphefumulelwe we-testosterone, izici zobuso ezingaphansi kwesifazane kanye nobubanzi obukhulu bekhanda kunezilawuli zabesifazane. Abesilisa abaseqenjini le-ASD bahlolwe njengabanezimpawu zomzimba zobudoda nezinga eliphansi lezwi, nezici zobuso ezi-androgynous ezihlanganiswe ngokuqinile nangokuqiniseka ngezimpawu ze-autistic ezilinganiswe ne-Autism-Spectrum Quotient kusampula ephelele.

Ababhali baphetha ngokuthi

Uma sihlanganisiwe, imiphumela yethu iphakamisa ukuthi abesifazane abane-ASD bakhuphule amazinga e-serum testosterone nokuthi, ezicini eziningana, bakhombisa izici zobudoda kakhulu kunabesifazane abangenayo i-ASD, kanti abesilisa abane-ASD bakhombisa izici zesifazane eziningi kunabesilisa abangenayo i-ASD. Esikhundleni sokuba yisifo esivezwa ubudoda kubo bobabili ubulili, i-ASD ngaleyo ndlela ibonakala iyisifo esidelela ubulili.

Ngokuqondile, ababhali baphawula lokho

Imiphumela yethu iyahambisana nombono wokuthi umthelela we-androgen ku-ASD uyathuthukiswa kwabesifazane kepha wehliswe emadodeni. Ngaphezu kwalokho, ocwaningweni lwezingane ezine-ASD kanye nobuphazamisi bobulili, cishe bonke kwakungabafana besilisa nabesifazane, kepha ngokusho kwe-androgen yokuqala yomthelela we-ASD, kufanele kulindeleke okuphikisayo. Ngakho-ke siguqula inkolelo-mbono kaBaron-Cohen, yokuthi i-autism kufanele ibhekwe njengomphumela wobudoda obedlulele bobuchopho, ngokusikisela ukuthi ingahle ihlotshaniswe nezici ze-androgynous kubo bobubili ubulili.

Nakulokhu futhi, umbono kaBaron-Cohen we-autism ubukeka sengathi uthinte umzimba. Ngempela, lokhu okutholakele kubonakala kuqinisekisa lokho kolunye ucwaningo lwakamuva oluphakamisa ukuthi ngokudabukisayo imfundiso eyedlulele yobuchopho besilisa isebenza kakhulu kwabesifazane kunabesilisa!

Ngokuya ngombono wobuchopho obhaliwe, lokhu okutholakele okuvusa inkanuko kumelela obunye ubufakazi obubalulekile bomqondo wezimbangela ze-epigenetic zesifo i-Asperger's syndrome eyabekwa kuqala ngo-2008 nguJulie R. Jones nabanye futhi yaphakanyiswa yimi ngokuzimela 2010.

Kanye nama-chromosomes angama-22 angewona owocansi (noma ama-autosomes, kwesobunxele) etholwe kumzali ngamunye, abesilisa bathola i-Y chromosome yocansi evela kubaba futhi i-X ivela kumama, kanti abesifazane bathola i-X kumzali ngamunye. Ukuze uvikele ukuphindwe kabili kwemikhiqizo yezakhi zofuzo i-X, iningi lezakhi zofuzo kwelinye lama-chromosomes amabili esifazane alisebenzi.

I-X chromosome inezakhi zofuzo ezingaba ngu-1500, okungenani eziyi-150 zazo ezihlobene nobuhlakani nezenhlalo, ukufunda ingqondo, noma amakhono okubonisa ukuqonda — engingakubiza ngokuthi ingqondo. Amawele esifazane afanayo ayahluka kakhulu ezilinganisweni zokuziphatha komphakathi kanye nekhono lokukhuluma uma kuqhathaniswa namawele wesilisa afanayo ngenxa yokwehlukaniswa kokungasebenzi kwe-X kwalezi zinhlayiya ezibalulekile zengqondo- isici se-epigenetic esiphikisana nokuhlakanipha okuvamile kokuthi noma yikuphi umehluko phakathi kwamawele afanayo kumele kube ngumphumela wokungafani -izakhi, imiphumela yemvelo.

Omaki be-epigenetic bomama ku-X owesifazane adlulela ezinganeni zakhe ngokuvamile bayasulwa, ukuze i-X isethwe kabusha ibe yi-epigenetically ibe ngu-zero. Kodwa lokhu akwenzeki njalo. Ngokuphambene nalokho, kokuthunyelwe kwami ​​kwasekuqaleni, ngiphakamise ukuthi ukugcinwa ngengozi kokungasebenzi kwezakhi zofuzo ezisemqoka ku-X umama azedlulisela endodaneni kungachaza kokubili ukusilela kwengqondo yendodana nokuvelela kwamacala wesilisa ka-Asperger (amadodakazi kunjalo ukuba kokukhulu okuvikelwe ngokuba nama-X amabili).

I-Asperger's Syndrome Essential Reads

Iseluleko Somshado Samahhala Esivela Kubantu Abadala baka-Asperger